Analyzing Virus Theory

The Coronavirus Hoax papers ( http://mileswmathis.com/corona.pdf and http://mileswmathis.com/cor2.pdf ) are the inspiration for this analysis.

NATURE'S PURPOSE FOR VIRUSES?
_If there were no decay, the biosphere would run out of organic matter on which to live, so all living things would die and the planet would be full of non-decaying dead bodies.
_My questions is: Do viruses attack living cells, or are viruses part of the decay process, or what?
_A lysosome is an organelle in the cytoplasm of eukaryotic cells (cells that contain a distinct membrane-bound nucleus) containing degradative enzymes enclosed in a membrane. When a cell is damaged and the membrane is pierced, the lysosome enzymes break down the cell for the body to recycle or remove. Are viruses decay products of cellular breakdown?

CANCER VIRUSES
_James Sloane used to have a forum at curezone.com called Truth in Medicine. He had worked in hospitals for 13 years as a med tech, I think. He quit because he knew of natural cures, but wasn't allowed to promote or use them there. On his forum he said most cancer is caused by viruses.

HIV VIRUS
_He said the HIV virus does not cause AIDS or anything. Dr. Gallo discovered the virus and claimed it caused AIDS, and the medical authorities went along with it, but apparently only because they could all make a lot of money testing for HIV antibodies and then using deadly drugs to treat it and blame the virus instead of the drugs when patients die.

I'm working on the issue of the coronavirus at https://futureschool.boards.net/post/44/thread

I don't get much time to work on this except mostly on weekends.

INFECTIOUS DOSE
I just learned that "infectious dose" is said to be the amount of viruses etc that are needed to cause an infection. But I think it's very likely that it depends on a person's state of health. A person with poor nutrition and lifestyle habits is likely to be much more prone to the infection than a healthy person.

IMMUNITY
Specifically, I think if the person's immune system is strong, viruses will be easily handled and won't be able to infect the person's cells.

VIRAL ENTRY
Viruses can enter the body primarily via the respiratory system and the alimentary canal.

RESPIRATORY SYSTEM
The respiratory system includes the: Nose and nasal cavity; Sinuses; Mouth; Throat (pharynx); Voice box (larynx); Windpipe (trachea); Diaphragm; Lungs; Bronchial tubes/bronchi; Bronchioles; Air sacs (alveoli); Capillaries.

ALIMENTARY CANAL
The alimentary canal is the whole passage along which food passes through the body from mouth to anus. It includes the esophagus, stomach, and intestines.

MUCOSA
A mucous membrane or mucosa is a membrane that lines various cavities in the body and covers the surface of internal organs. It consists of one or more layers of epithelial cells overlying a layer of loose connective tissue. Mucous membranes line body cavities and canals that lead to the outside, chiefly the respiratory, digestive, and urogenital tracts. Mucous membranes line many tracts and structures of the body, including the mouth, nose, eyelids, trachea (windpipe) and lungs, stomach and intestines, and the ureters, urethra, and urinary bladder.

MUCOSAL IMMUNE SYSTEM
The mucosal immune system consists of organized and dispersed lymphoid tissues that are closely associated with mucosal epithelial surfaces, and mucosal immune responses generated in one location are transferred throughout the mucosal immune system by lymphocytes programmed to home to regional effector sites.

MUCOSAL IMMUNITY AND VIRAL INFECTIONS
https://www.ncbi.nlm.nih.gov/pubmed/11370770
Abstract
The mucosal surfaces are the first portals of entry for most infectious agents, among which respiratory and intestinal viruses are of greatest epidemiological importance. To combat these infections, the immune system uses unspecific and specific mechanisms. Unspecific responses include the production of virus-induced cytokines, such as type 1 interferons and natural killer (NK) cell activity, while specific immune responses mainly depend on cytotoxic T cells, which are important especially in the early course of a viral infection, and on antibodies. At the mucosal sites, antiviral secretory IgA antibodies play a major role in clearing viral infections and preventing or modifying disease after re-exposure. Passive transfer of virus-specific antibodies has been used in experimental and clinical settings to prevent or treat viral mucosal infections. [The following sentence is surely false.] In the future, the development of new mucosal vaccines promises to have the strongest impact on the epidemiology of viral infections. [No, improved nutrition would have the greatest positive impact.]

VACCINATION
Vaccination is pretended to be a means to destroy viruses and other pathogens in the body, but in reality it has proved to be another means for viruses to enter the body. Some of the polio vaccines were found to be contaminated with such viruses as SV40 (simian virus 40 from monkeys), which infected millions of "boomers" of the older generation and often causes brain cancer. As James Sloane stated, vaccines have never been tested for safety or effectiveness. Neil Z. Miller wrote a book on that.

NCBI (PUBMED) STUDIES

(These following articles may be mostly useless, but I wanted to see if there's any good info in any of them.)

1. High-resolution computed tomography manifestations of COVID-19 infections in patients of different ages.
https://www.ncbi.nlm.nih.gov/pubmed/32240913
Abstract
PURPOSE:
We aimed to compare chest HRCT lung signs identified in scans of differently aged patients with COVID-19 infections.
METHODS:
Case data of patients diagnosed with COVID-19 infection in Hangzhou City, Zhejiang Province in China were collected, and chest HRCT signs of infected patients in four age groups (<18 years, 18-44 years, 45-59 years, ≥60 years) were compared.
RESULTS:
Small patchy, ground-glass opacity (GGO), and consolidations were the main HRCT signs in 98 patients with confirmed COVID-19 infections. Patients aged 45-59 years and aged ≥60 years had more bilateral lung, lung lobe, and lung field involvement, and greater lesion numbers than patients <18 years. GGO accompanied with the interlobular septa thickening or a crazy-paving pattern, consolidation, and air bronchogram sign were more common in patients aged 45-59 years, and ≥60 years, than in those aged <18 years, and aged 18-44 years.
CONCLUSIONS:
Chest HRCT manifestations in patients with COVID-19 are related to patient's age, and HRCT signs may be milder in younger patients.
Copyright 2020 Elsevier B.V. All rights reserved.

2. Nervous system involvement after infection with COVID-19 and other coronaviruses.
https://www.ncbi.nlm.nih.gov/pubmed/32240762
Abstract
Viral infections have detrimental impacts on neurological functions, and even to cause severe neurological damage. Very recently, coronaviruses (CoV), especially severe acute respiratory syndrome CoV 2 (SARS-CoV-2), exhibit neurotropic properties and may also cause neurological diseases. It is reported that CoV can be found in the brain or cerebrospinal fluid. The pathobiology of these neuroinvasive viruses is still incompletely known, and it is therefore important to explore the impact of CoV infections on the nervous system. Here, we review the research into neurological complications in CoV infections and the possible mechanisms of damage to the nervous system.
Copyright 2020. Published by Elsevier Inc.

3. Coronavirus Disease 2019 in elderly patients: characteristics and prognostic factors based on 4-week follow-up.
https://www.ncbi.nlm.nih.gov/pubmed/32240670
Abstract
OBJECTIVE:
To investigate the characteristics and prognostic factors in the elderly patients with COVID-19.
METHODS:
Consecutive cases over 60 years old with COVID-19 in Renmin Hospital of Wuhan University from Jan 1 to Feb 6, 2020 were included. The primary outcomes were death and survival till March 5. Data of demographics, clinical features, comorbidities, laboratory tests and complications were collected and compared for different outcomes. Cox regression was performed for prognostic factors.
RESULTS:
339 patients with COVID-19 (aged 71±8 years,173 females (51%)) were enrolled, including 80 (23.6%) critical, 159 severe (46.9%) and 100 moderate (29.5%) cases. Common comorbidities were hypertension (40.8%), diabetes (16.0%) and cardiovascular disease (15.7%). Common symptoms included fever (92.0%), cough (53.0%), dyspnea (40.8%) and fatigue (39.9%). Lymphocytopenia was a common laboratory finding (63.2%). Common complications included bacterial infection (42.8%), liver enzyme abnormalities (28.7%) and acute respiratory distress syndrome (21.0%). Till Mar 5, 2020, 91 cases were discharged (26.8%), 183 cases stayed in hospital (54.0%) and 65 cases (19.2%) were dead. Shorter length of stay was found for the dead compared with the survivors (5 (3-8 ) vs. 28 (26-29), P < 0.001). Symptoms of dyspnea (HR 2.35, P = 0.001), comorbidities including cardiovascular disease (HR 1.86, P = 0.031) and chronic obstructive pulmonary disease (HR 2.24, P = 0.023), and acute respiratory distress syndrome (HR 29.33, P < 0.001) were strong predictors of death. And a high level of lymphocytes was predictive of better outcome (OR = 0.10, P < 0.001).
CONCLUSIONS:
High proportion of severe to critical cases and high fatality rate were observed in the elderly COVID-19 patients. Rapid disease progress was noted in the dead with a median survival time of 5 days after admission. Dyspnea, lymphocytopenia, comorbidities including cardiovascular disease and chronic obstructive pulmonary disease, and acute respiratory distress syndrome were predictive of poor outcome. Close monitoring and timely treatment should be performed for the elderly patients at high risk.

4. Estimates of the severity of coronavirus disease 2019: a model-based analysis.
https://www.ncbi.nlm.nih.gov/pubmed/32240634
Abstract
BACKGROUND:
In the face of rapidly changing data, a range of case fatality ratio estimates for coronavirus disease 2019 (COVID-19) have been produced that differ substantially in magnitude. We aimed to provide robust estimates, accounting for censoring and ascertainment biases.
METHODS:
We collected individual-case data for patients who died from COVID-19 in Hubei, mainland China (reported by national and provincial health commissions to Feb 8, 2020), and for cases outside of mainland China (from government or ministry of health websites and media reports for 37 countries, as well as Hong Kong and Macau, until Feb 25, 2020). These individual-case data were used to estimate the time between onset of symptoms and outcome (death or discharge from hospital). We next obtained age-stratified estimates of the case fatality ratio by relating the aggregate distribution of cases to the observed cumulative deaths in China, assuming a constant attack rate by age and adjusting for demography and age-based and location-based under-ascertainment. We also estimated the case fatality ratio from individual line-list data on 1334 cases identified outside of mainland China. Using data on the prevalence of PCR-confirmed cases in international residents repatriated from China, we obtained age-stratified estimates of the infection fatality ratio. Furthermore, data on age-stratified severity in a subset of 3665 cases from China were used to estimate the proportion of infected individuals who are likely to require hospitalisation.
FINDINGS:
Using data on 24 deaths that occurred in mainland China and 165 recoveries outside of China, we estimated the mean duration from onset of symptoms to death to be 17·8 days (95% credible interval [CrI] 16·9-19·2) and to hospital discharge to be 24·7 days (22·9-28·1). In all laboratory confirmed and clinically diagnosed cases from mainland China (n=70 117), we estimated a crude case fatality ratio (adjusted for censoring) of 3·67% (95% CrI 3·56-3·80). However, after further adjusting for demography and under-ascertainment, we obtained a best estimate of the case fatality ratio in China of 1·38% (1·23-1·53), with substantially higher ratios in older age groups (0·32% [0·27-0·38] in those aged <60 years vs 6·4% [5·7-7·2] in those aged ≥60 years), up to 13·4% (11·2-15·9) in those aged 80 years or older. Estimates of case fatality ratio from international cases stratified by age were consistent with those from China (parametric estimate 1·4% [0·4-3·5] in those aged <60 years [n=360] and 4·5% [1·8-11·1] in those aged ≥60 years [n=151]). Our estimated overall infection fatality ratio for China was 0·66% (0·39-1·33), with an increasing profile with age. Similarly, estimates of the proportion of infected individuals likely to be hospitalised increased with age up to a maximum of 18·4% (11·0-7·6) in those aged 80 years or older.
INTERPRETATION:
These early estimates give an indication of the fatality ratio across the spectrum of COVID-19 disease and show a strong age gradient in risk of death.
FUNDING:
UK Medical Research Council.

5. First Mildly Ill, Non-Hospitalized Case of Coronavirus Disease 2019 (COVID-19) Without Viral Transmission in the United States - Maricopa County, Arizona, 2020.
https://www.ncbi.nlm.nih.gov/pubmed/32240285
Abstract
BACKGROUND:
Coronavirus disease 2019 (COVID-19) causes a range of illness severity. Mild illness has been reported, but whether illness severity correlates with infectivity is unknown. We describe the public health investigation of a mildly ill, non-hospitalized COVID-19 case who traveled to China.
METHODS:
The case was a Maricopa County resident with multiple severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2)-positive specimens collected on January 22, 2020. Contacts were persons exposed to the case on or after the day before case diagnostic specimen collection. Contacts were monitored for 14 days after last known exposure. High-risk contacts had close, prolonged case contact (≥10 minutes within 2 meters). Medium-risk contacts wore all U.S. Centers for Disease Control and Prevention (CDC)-recommended personal protective equipment during interactions. Nasopharyngeal and oropharyngeal (NP/OP) specimens were collected from the case and high-risk contacts and tested for SARS-CoV-2.
RESULTS:
Paired case NP/OP specimens were collected for SARS-CoV-2 testing at 11 time points. In 8 pairs (73%), ≥1 specimen tested positive or indeterminate, and in 3 pairs (27%) both tested negative. Specimens collected 18 days after diagnosis tested positive. Sixteen contacts were identified; 11 (69%) had high-risk exposure, including 1 intimate contact, and 5 (31%) had medium-risk exposure. In total, 35 high-risk contact NP/OP specimens were collected for SARS-CoV-2 testing; all 35 pairs (100%) tested negative.
CONCLUSIONS:
This report demonstrates that SARS-CoV-2 infection can cause mild illness and result in positive tests for up to 18 days after diagnosis, without evidence of transmission to close contacts. These data might inform public health strategies to manage individuals with asymptomatic infection or mild illness.
Published by Oxford University Press for the Infectious Diseases Society of America 2020. This work is written by (a) US Government employee(s) and is in the public domain in the US.

6. Post-donation COVID-19 identification in blood donors.
https://www.ncbi.nlm.nih.gov/pubmed/32240537
Abstract
The coronavirus disease 19 (COVID-19) outbreak, which was characterized as a pandemic on 11 March 2020 by the WHO, started in December 2019 with the emergence of pneumonia cases of unknown cause in Wuhan, Hubei, China [1]. SARS-CoV-2, the causative agent of COVID-19, are enveloped, non-segmented, single stranded positive sense RNA viruses and are classified as a sister clade to the prototype human and bat severe acute respiratory syndrome coronaviruses (SARS-CoVs) of the species Severe acute respiratory syndrome-related coronavirus [2]. So far, no respiratory virus, including SARS-CoV and the Middle East Respiratory Syndrome (MERS)-CoV, has been confirmed as transfusion-transmissible [3,4].

7. Isolated sudden onset anosmia in COVID-19 infection. A novel syndrome?
[anosmia is the loss of the sense of smell, either total or partial. It may be caused by head injury, infection, or blockage of the nose.]
https://www.ncbi.nlm.nih.gov/pubmed/32240279
Abstract
BACKGROUND:
The amelioration of the current COVID pandemic relies on swift and efficient case finding as well as stringent social distancing measures. Current advice suggests that fever or new onset dry cough are the commonest presenting complaints.
METHODOLOGY:
We present a case report and case series as well as other evidence that there is an important fourth presenting syndrome, namely isolated sudden onset anosmia (ISOA), which should be considered highly suspicious for SARS-CoV-2.
RESULTS:
A patient presenting with ISOA who went on to test positive for infection with COVID-19 and did not develop any further symptoms as well as a case series of similar patients although limited by the lack of reliable testing at the moment.
CONCLUSIONS:
We posit the existence of a fourth common syndrome of COVID-19 infection: isolated sudden onset anosmia (ISOA) and urge the international community to consider this presentation in current management advice.

8. Preliminary Estimates of the Prevalence of Selected Underlying Health Conditions Among Patients with Coronavirus Disease 2019 - United States, February 12-March 28, 2020.
CDC COVID-19 Response Team.
https://www.ncbi.nlm.nih.gov/pubmed/32240123
Abstract
On March 11, 2020, the World Health Organization declared Coronavirus Disease 2019 (COVID-19) a pandemic (1). As of March 28, 2020, a total of 571,678 confirmed COVID-19 cases and 26,494 deaths have been reported worldwide (2). Reports from China and Italy suggest that risk factors for severe disease include older age and the presence of at least one of several underlying health conditions (3,4). U.S. older adults, including those aged ≥65 years and particularly those aged ≥85 years, also appear to be at higher risk for severe COVID-19-associated outcomes; however, data describing underlying health conditions among U.S. COVID-19 patients have not yet been reported (5). As of March 28, 2020, U.S. states and territories have reported 122,653 U.S. COVID-19 cases to CDC, including 7,162 (5.8%) for whom data on underlying health conditions and other known risk factors for severe outcomes from respiratory infections were reported. Among these 7,162 cases, 2,692 (37.6%) patients had one or more underlying health condition or risk factor, and 4,470 (62.4%) had none of these conditions reported. The percentage of COVID-19 patients with at least one underlying health condition or risk factor was higher among those requiring intensive care unit (ICU) admission (358 of 457, 78%) and those requiring hospitalization without ICU admission (732 of 1,037, 71%) than that among those who were not hospitalized (1,388 of 5,143, 27%). The most commonly reported conditions were diabetes mellitus, chronic lung disease, and cardiovascular disease. These preliminary findings suggest that in the United States, persons with underlying health conditions or other recognized risk factors for severe outcomes from respiratory infections appear to be at a higher risk for severe disease from COVID-19 than are persons without these conditions.

9. COVID-19 Outbreak Associated with Air Conditioning in Restaurant, Guangzhou, China, 2020.
https://www.ncbi.nlm.nih.gov/pubmed/32240078
Abstract
During January 26-February 10, 2020, an outbreak of 2019 novel coronavirus disease in an air-conditioned restaurant in Guangzhou, China, involved 3 family clusters. The airflow direction was consistent with droplet transmission. To prevent the spread of the virus in restaurants, we recommend increasing the distance between tables and improving ventilation.

10. Clinical characteristics of Non-ICU hospitalized patients with coronavirus disease 2019 and liver injury：A Retrospective study.
https://www.ncbi.nlm.nih.gov/pubmed/32239591
Abstract
BACKGROUND & AIMS:
Coronavirus disease 2019 (COVID-19) has raised world concern for global epidemic since December, 2019. Limited data are available for liver function in COVID-19 patients. We aimed to investigate the risk factors related to liver injury in the COVID-19 patients.
METHODS:
A retrospective study was performed in Non-ICU Ward at Jinyintan Hospital from February 2, 2020 to February 23, 2020. Consecutively confirmed COVID-19 discharged cases were enrolled. The clinical characteristics of patients with liver injury and without liver injury were compared.
RESULTS:
A total of 79 COVID-19 patients were included. 31.6%, 35.4% and 5.1% COVID-19 patients had elevated levels of ALT, AST and bilirubin, respectively. Median value of ALT, AST and bilirubin for entire cohort were 36.5 (17.5~71.5) U/L, 34.5 (25.3~ 55.3) U/L and 12.7 (8.1~15.4) mmol/L, respectively. There were no significant differences in age, previous medical history, and symptoms between the two groups. Males were more likely to have liver injury when infected with COVID-19 (p<0.05); Compared to patients without liver injury, patients with liver injury had increased levels of white blood cell counts, neutrophils, CRP and CT score (p< 0.05), and had a longer length of stay (p< 0.05). Logistic regression analyses suggested that the extent of pulmonary lesions on CT was a predictor of liver function damage (p< 0.05).
CONCLUSIONS:
Liver injury is common in Non-ICU hospitalized COVID-19 patients. It may be related to systemic inflammation. Intense monitoring and evaluation of liver function in patients with severe pulmonary imaging lesions should be considered.
This article is protected by copyright. All rights reserved.

11. Elevated plasmin(ogen) as a common risk factor for COVID-19 susceptibility.
https://www.ncbi.nlm.nih.gov/pubmed/32216698
Abstract
Patients with hypertension, diabetes, coronary heart disease, cerebrovascular illness, COPD, and kidney dysfunction have worse clinical outcomes when infected with SARS-CoV-2, for unknown reasons. The purpose of this review is to summarize the evidence for the existence of elevated plasmin(ogen) in COVID-19 patients with these comorbid conditions. Plasmin, and other proteases, may cleave a newly inserted furin site in the S protein of SARS-CoV-2, extracellularly, which increases its infectivity and virulence. Hyper-fibrinolysis associated with plasmin leads to elevated D-dimer in severe patients. The plasmin(ogen) system may prove a promising therapeutic target for combating COVID-19.

12. The neuroinvasive potential of SARS-CoV2 may play a role in the respiratory failure of COVID-19 patients.
https://www.ncbi.nlm.nih.gov/pubmed/32104915
Abstract
Following the severe acute respiratory syndrome coronavirus (SARS-CoV) and Middle East respiratory syndrome coronavirus (MERS-CoV), another highly pathogenic coronavirus named SARS-CoV-2 (previously known as 2019-nCoV) emerged in December 2019 in Wuhan, China, and rapidly spreads around the world. This virus shares highly homological sequence with SARS-CoV, and causes acute, highly lethal pneumonia coronavirus disease 2019 (COVID-19) with clinical symptoms similar to those reported for SARS-CoV and MERS-CoV. The most characteristic symptom of patients with COVID-19 is respiratory distress, and most of the patients admitted to the intensive care could not breathe spontaneously. Additionally, some patients with COVID-19 also showed neurologic signs, such as headache, nausea, and vomiting. Increasing evidence shows that coronaviruses are not always confined to the respiratory tract and that they may also invade the central nervous system inducing neurological diseases. The infection of SARS-CoV has been reported in the brains from both patients and experimental animals, where the brainstem was heavily infected. Furthermore, some coronaviruses have been demonstrated able to spread via a synapse-connected route to the medullary cardiorespiratory center from the mechanoreceptors and chemoreceptors in the lung and lower respiratory airways. Considering the high similarity between SARS-CoV and SARS-CoV2, it remains to make clear whether the potential invasion of SARS-CoV2 is partially responsible for the acute respiratory failure of patients with COVID-19. Awareness of this may have a guiding significance for the prevention and treatment of the SARS-CoV-2-induced respiratory failure.

DATA

German Infectologist's Open Letter to Merkel Re Government Overreaction
https://www.shtfplan.com/emergency-preparedness/german-infectologist-decimates-covid-19-doomsday-cult-in-open-letter-to-merkel_04022020
A number of coronaviruses have been circulating for a long time – largely unnoticed by the media. If it should turn out that the COVID-19 virus should not be ascribed a significantly higher risk potential than the already circulating corona viruses, all countermeasures would obviously become unnecessary.
The internationally recognized International Journal of Antimicrobial Agents will soon publish a paper that addresses exactly this question. Preliminary results of the study can already be seen today and lead to the conclusion that the new virus is NOT different from traditional corona viruses in terms of dangerousness. The authors express this in the title of their paper „SARS-CoV-2: Fear versus Data“.

SARS-CoV-2: fear versus data.
Int J Antimicrob Agents. 2020 Mar 19
https://www.ncbi.nlm.nih.gov/pubmed/32201354
Abstract
SARS-CoV-2, the novel coronavirus from China, is spreading around the world, causing a huge reaction despite its current low incidence outside China and the Far East. Four common coronaviruses are in current circulation and cause millions of cases worldwide. This article compares the incidence and mortality rates of these four common coronaviruses with those of SARS-COV-2 in Organisation for Economic Co-operation and Development countries [Europe: i.e. Austria, Belgium, Czech Republic, Denmark, Estonia, Finland, France, Germany, Greece, Hungary, Iceland, Ireland, Italy, Latvia, Luxembourg, Netherlands, Norway, Poland, Portugal, Slovak Republic, Slovenia, Spain, Sweden, Switzerland, and the United Kingdom.]. It is concluded that the problem of SARS-CoV-2 is probably being overestimated, as 2.6 million people die of respiratory infections each year compared with less than 4000 deaths for SARS-CoV-2 at the time of writing.

406

Virology and Vaccine Facts | Why Coronavirus Is Not Contagious
https://ourgreaterdestiny.org/2020/04/emergency-hearing-c-o-v-i-d-and-5g-vir0l0gy-facts/

I'd like to compare that claim with James Sloane's claim that specific viruses can cause cancer etc.

I want to edit the following material if I get time.

-----

OurGreaterDestiny
New World Consciousness [NWC]

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← Emergency 5G Coronavirus Hearing | Vir0l0gy Facts reposted
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Emergency Hearing C O V I D and 5G + Vir0l0gy Facts
Posted on April 8, 2020 by ourgreaterdestiny
I N T J
Due to skewed text this is a re-post of https://ourgreaterdestiny.org/2020/04/emergency-5g-coronavirus-hearing-vir0l0gy-facts/
I sense one or more of the following individuals is controlled opposition however the facts are vital so please share.

Louis Pasteur Vs Antoine Béchamp and The Germ Theory of Disease Causation
We do not catch diseases. We build them. We have to eat, drink, think, and feel them into existence. We work hard at developing our diseases. We must work just as hard at restoring health. The presence of germs does not constitute the presence of a disease. Bacteria are scavengers of nature…they reduce dead tissue to its smallest element. Germs or bacteria have no influence, whatsoever, on live cells. Germs or microbes flourish as scavengers at the site of disease. They are just living on the unprocessed metabolic waste and diseased, malnourished, nonresistant tissue in the first place. They are not the cause of the disease, any more than flies and maggots cause garbage. Flies, maggots, and rats do not cause garbage but rather feed on it. Mosquitoes do not cause a pond to become stagnant! You always see firemen at burning buildings, but that doesn’t mean they caused the fire…
Traditional Western medicine teaches and practices the doctrines of French chemist Louis Pasteur (1822-1895). Pasteur’s main theory is known as the Germ Theory Of Disease. It claims that fixed species of microbes from an external source invade the body and are the first cause of infectious disease. The concept of specific, unchanging types of bacteria causing specific diseases became officially accepted as the foundation of allopathic Western medicine and microbiology in late 19th century Europe. Also called monomorphism, (one-form), it was adopted by America’s medical/industrial complex, which began to take shape near the turn of the century. This cartel became organized around the American Medical Association, formed by drug interests for the purpose of manipulating the legal system to destroy the homeopathic medical profession.
Controlled by pharmaceutical companies, the complex has become a trillion-dollar-a-year business. It also includes many insurance companies, the Food and Drug Administration (FDA), the National Institutes of Health (NIH), the Centers for Disease Control (CDC), hospitals, and university research facilities. The microbian doctrine gave birth to the technique of vaccination that was blindly begun in 1796 by Edward Jenner. Jenner took pus from the running sores of sick cows and injected it into the blood of his “patients.” Thus was born a vile practice (immunization/vaccination) whose nature has changed little to this day, and whose understanding is still clouded by Pasteur’s theory. This also gave birth to the development of antibiotics, the first being penicillin in 1940. An antibiotic is the poisonous waste from one germ used in the attempt to kill another. Penicillin is the poison from a fungus. This has created the proliferation of aggressive and stubborn forms of resistant strains that haunt us today. Read more at http://www.laleva.org/eng/2004/05/louis_pasteur_vs_antoine_bchamp_and_the_germ_theory_of_disease_causation_-_1.html
Virology and Vaccine Facts | Why Coronavirus Is Not Contagious
The following anonymous post on the true nature of viruses and virology nails to the mast, fraudulent claims used to justify the indefensible.
This post intends to go to the heart of virology and vaccination and what vaccination is propped up by – the entirety of virology and vaccine science is predicated on one thing—that viruses are infectious agents that cause disease. Without this theory, vaccines would not be ‘effective’ or ‘work’ in the minds of the people. Without the virus theory, vaccines would crumble like a house of cards.
This article shows the pseudoscience behind theories that prop up virology and the vast problems with it.
Viruses Are Not Living Organisms
Firstly, viruses are not living organisms or living microbes. They do not have a respiratory system, nor do they have a nucleus or digestive system.
Viruses are not alive and viruses are not contagious.
The fear behind Coronavirus, for instance, is wholly unwarranted.
Forget everything you think you know about viruses and bacteria. You have been lied to.
​The science of virology is based upon the study of viruses. However, no real footage of viral activity exists (except for a recently released (2018) short footage of an HIV virus which shows merely 20% of the virus theory process). Such footage is merely 3D animation and models.
Scientific Encyclopedia states viruses have been obtained for experimentation by means of extremely powerful centrifuges which must be specially built.
Viruses are so small that they average around 0.1 microns in size for a typical virus.​
Observation Of Viruses Is Inherently Flawed
Viruses are observed in cell cultures/petri-dish environments.
Cell cultures are grown in controlled conditions outside their natural environment, wherein cells are artificially kept alive by fluids that are toxic and do damage to cellular activity.
In such a sterile environment, cells cannot utilize the full range of their normal cleansing methods as they would in the human body.
​Those processes are:
Phagocytosis (and all of its processes)
Bacterial
Fungal
Parasitical
Viral (virus)
In the processes of phagocytosis, cellular debris and dead and dying tissue are absorbed and discarded for elimination out of the body.
It is bacteria that first and foremost carry out this process in large part—mainly as scavengers.
Fungus and parasites are called upon as needed in special cases, and in this process, small amounts of viruses may be utilized to accompany all other processes.
All of these processes are alive, but viruses are not alive.
In such an artificial environment wherein cells are kept alive but not healthy by serums, cells will degenerate, and their viral janitors will become prominent.
Viruses do not multiply on their own. When added to fertile petri-dishes that sustain cellular life, no additional viral protein structures appear.
Only when cells are added is there multiplication of viral protein structures. However, this is because petri-dishes are not the proper or healthy environment for cells, and so viral waste occurs.
This is because cells must manufacture viruses to cleanse themselves in such a toxic environment since they do not have access to the full range of their cleansing processes as would occur in the body. I will show why—
Note: Viruses are necessary to dissolve dead and dying tissue when tissue is so toxic that living microbes cannot feed upon and eliminate those tissues, waste, and cellular debris without being poisoned to death.
​When Would Viral Activity Become Prominent?
​As stated, viruses may accompany these processes in small amounts. However, viruses will only become prominent when all these other processes have been largely killed due to:
Environmental toxicity
Pollution
Chemical inundation
Poor air quality
Poor water quality
Poor food quality
Nutritional deficiencies
Wrong combination or choice of foods
Medical treatment, such as antibiotics and medications
When a body has a high degree of toxicity, bacteria feeding upon that toxic dead matter and tissue will be poisoned to death.
When the body is at such a point of systemic toxicity, where bacterial levels and all living microbes in the body have been diminished or killed due to the above reasons, the body will call upon the help of viruses to help cleanse itself.
When the body cannot utilize milder methods, such as a cold (usually bacterial), it will utilize the help of non-living protein solvents which are known as viruses. I will show why this is the only logical answer.
​Viruses help consume and eliminate substances into small particles that can then be expelled via mucous membranes, out through the skin, or through the intestinal tract.
Cells produce viruses when their tissues are so toxic that phagocytes, parasites, bacteria, and fungi cannot help cleanse, repair and regenerate their tissues and fluids.
Science states, incorrectly without proof, that viruses originate outside the body, then ‘hijack’ the RNA or DNA of the cell, and then replicate whilst attacking cells indiscriminately. If this were true, viruses would replicate endlessly, eventually attacking all healthy cells, but they do not.
We know that antibodies, a type of white blood cell, regulates the virus.
There exists no video evidence of viruses hijacking cells, except for 3D renders, and animations based on theory.
​The True Creation of Viruses (simplistic view)
Science falsely claims that viruses replicate themselves. In reality, it is the cell itself that is producing the virus.
Notice how viruses are manufactured by a healthy cell but do not destroy it.
RNA and/or DNA is given by the host cell to dissolve specific substances within the body. If this were not the case, the virus would destroy the cell which created it, but it does not.
The virus is ejected, damaging part of the cell, but not destroying it completely. The cell is then able to repair itself in time.
Cells conspire as one unit to cleanse themselves and their surroundings so that new cellular activity can thrive.
Large amounts of viral activity are present when the body is unable to use milder living microbial detoxification methods to cleanse itself due to systemic toxicity of tissues that poison living microbes.
​Steps for Creation of a Virus
Viral proteins part of the genome of the living body existing in every cell which determines what type of proteins will be created by a cell is called into action.
Viral proteins existing in the cell enter the nucleus of the cell. Viruses are manufactured in their whole form within the cell, and sequenced/encoded via RNA/DNA host directives.
The virus leaves the nucleus and is housed in the cell until it leaves the cell.
The virus is ejected by the cell, damaging a part of the cell, but not destroying it.
Viruses change every 72 hours.
Virus replication continues and every 72 hours the first strain is exhausted and an entirely new set of viruses is then manufactured by cells to continue the job of the previous, until the process is complete.
How Viruses are Manufactured | The True Processes of the Virus
​Viruses do not infect healthy stable cells. They dissolve dead and decaying cells and tissue, dissolving them so that new cellular activity can thrive.
A good analogy:
Flies appear on dead matter but are not the cause of the dead matter. They are scavengers that break down dead matter. In this way, viruses and bacteria operate in the same exact manner within the body. Without scavengers on Earth to clean up waste, Earth’s air would become toxic. The same processes are carried out in the body on a microscopic macro level.
Science states the opposite of what reality dictates to us through our own observation of nature. This is impossible because our bodies are microcosms for the way nature operates outside our bodies. Assuming the opposite of this goes against our observable nature and is foolish.
​As stated, when the normal janitorial functions of the body have been largely diminished and killed due to systemic toxicity, cells can no longer maintain themselves. Red blood cells come together as a whole unit to save themselves and conspire to cleanse themselves by manufacturing solvent protein constructs (virus) that disassemble and break down dead and dying cells, cellular waste, tissue, and foreign debris.
Cells manufacture viruses in their whole form cellularly. In this process, viruses are manufactured directly within the cell using pre-existing viral protein in the cell and genome, and are embedded/encoded with RNA and/or DNA by the host cell.
The cell ejects the virus, which is then regulated by white blood cells through that encoding (antibodies), which oversee the processes of the virus. This allows the viral activity to be controlled and regulated properly.
These two functions are united as one process, and they do not act separately. Once the cell ejects this virus, the cell is partially damaged but is not destroyed. The viruses, which are many, consume and dissolve dead, dying and foreign tissue, debris, unhealthy cells, and cellular waste.
This process takes time depending on the toxicity involved. The effects of their elimination are the symptoms experienced in cold or flu. Viruses break these substances down into tiny particles that can then be expelled via mucous, skin, and bowels.
When the process is complete, the body becomes stronger, so long as that person does not continue to toxify his or her body further. If he or she does, such extreme detoxifications will always occur.
​Viral Facts
Viruses cannot enter through the skin or eyes. Such vectors do not work because the mucus membranes and the immune system discard small amounts of foreign proteins such as viruses.
Viruses cannot enter through wounds because we bleed outwardly, not inwardly.
Viruses do not ‘exist’ outside of petri-dish solutions or a living body.
Viruses cannot function without a host cell that manufactures them and encodes them, and viruses cannot replicate without a host cell.
Viruses do not ‘infect’ or ‘invade’ cells. They are not alive to do so in the first place.
Viruses almost never dissolve living tissue, unless in specific circumstances such as polio and degenerative nervous system diseases where metal toxicity is present.
Viruses’ primary function is to dissolve dead matter.
Cells produce different viral strains depending on the condition of the tissue involved.
There are 320,000 viral strains inherent to the human body, and each cell contains the viral protein makeup to manufacture each strain when the body calls for it.
Viruses are sequenced/encoded by blood cells via RNA/DNA to break down specific dead and dying tissue and waste.
Viruses are very specific protein structures.
Coughing, sneezing, and spitting is not a vector for the transmission of viruses. Saliva and mucus membranes break down any such particles.
Skin is not a vector either because viruses cannot cross dead skin layers.
Viruses are a result of internal toxicity caused by the environment.
Viruses are cyclical in animals.
Viruses feed upon waste products in the blood and tissue.
The only way to get a virus outside of natural means is via direct injection (vaccine) or blood transfusions of a patient who has a virus. However, in such cases, the body only analyzes it as foreign tissue that must be eliminated.
Since the virus did not originate within the bodily host, that body does not know the time and place that the virus will be active nor does it have the key to decode it (RNA or DNA encoded by the cell) and cannot find the time of its activity.
As such, it is analyzed as a foreign substance that must be eliminated. Protein solvents (viruses) are manufactured of varying strengths to discard this waste if living microbes cannot eliminate it.
Throughout the year, upon season and climatic/temperature changes, the body will dump mass amounts of toxins into the blood for removal. Some of these toxins are so toxic in nature, such as mercury, formaldehyde, and other chemical byproducts, that living microbes cannot feed upon and eliminate them without dying.
Non-living proteins are then manufactured by each cell in the corresponding location of the body where this cleansing is necessary. Those toxic substances are disassembled and broken down by viruses so that the body can eliminate them, restoring homeostasis.
The only way viruses can be used as biological weapons is via injection, period. It is possible that such man made viral strains are included in regular existing vaccines, and this should not be ruled out as a possibility, but as previously stated, viral strains from outside the body are not recognized.
However, man-made substances that are injected can be designed to provoke extreme reactions in humans via various levels of tissue sterilization and adjuvants.
Viruses cannot cross-species ie; from animal to mankind. It is impossible for humans to develop animal flus—A. Because viruses are not contagious, and, B. Because animal RNA/DNA is not compatible with human RNA/DNA.
The only way animal tissue can be observed in the blood is through injection of animal tissues, which make their way to the blood, bypassing the digestive tract. Only then will swine tissue, or bird tissue, or any such animal tissue appear in the body.
When animal meat is consumed by a human, it is converted into human tissue. Human cells cannot produce animal cells or viruses. If we develop viruses, they are human viruses. Even if animal viruses ‘hijacked’ human cells, human cells cannot possibly produce animal viruses
Coronavirus is a respiratory virus manufactured by cells in the lungs and respiratory areas to cleanse themselves of systemic toxicity.
Such a cold virus occurs and functions in 4 main steps listed below.
Coughing brings blood and nutrients to the respiratory system. The symptoms associated with their removal are what occurs during SARS. Such airborne toxic substances are caused by burning plastics, formaldehyde, and factory tainted air, which encompasses a wide array of very toxic byproducts.
Older individuals with already weakened immune systems are prone to more advanced respiratory virus detoxifications and will account for most deaths. This illness may crop up in millions due to dense populations like in China breathing in such air on a daily basis. This does not mean it is contagious—it’s not.
​The 4 Main Steps For Coronavirus Creation
​1. Chemically toxic substances from the air are breathed into the lungs and respiratory system.
Toxic particles land onto the surface of the lungs and the fluid-filled sacs in the lungs (alveoli), where they cannot be dislodged or dissolved by living microbes because of their toxicity and nature.
Specific non-living protein solvent structures (virus) are then manufactured by cells in the respiratory system to disassemble and break down these substances in the lungs.
Mild flu-like symptoms usually result, including coughing and fever, which initiate the cleansing and healing process.
Why Viruses Arise In The Body
As previously stated, the processes of phagocytosis, fungal, parasitical, and bacterial, which are all living microbes, are responsible for consuming and eliminating dead cells, cellular waste, and foreign debris. But when tissue is so toxic that those living microbes cannot feed upon and eliminate those substances without being poisoned to death, cells will conspire to cleanse themselves by manufacturing specific non-living solvents know as viruses, which break down and disassemble those substances into particles to be expelled out through the skin, mucus, and bowels.
Viruses leave the cell, damaging only a part of the cell, but not destroying it. Once out of the cell, they are regulated by white blood cell antibodies to dissolve specific tissues and debris necessary to restore relative homeostasis.
Viruses do not destroy the cell wherein they are replicated, yet science states they infect other cells and DO destroy other cells indiscriminately, which has no proof and makes no logical sense. Such a theory is obviously untrue because then viruses would attack every living cell without a cause, killing the body every time, but this does not happen. Viruses only dissolve dead and dying waste in almost all circumstances.
The only time a virus would appear to attack living tissue is when metals are embedded in the tissue, such as polio cases, where viruses have to get into spinal column areas and cleanse tissue. Since metal is hard to remove from the body, it is natural for viruses to break down living tissue to remove those metals, which gives the illusion that the virus is somehow working against the body. In reality, the virus is attempting to heal the systemic toxicity of the body and reverse it.
​Conclusion
There is no other explanation for how the human body maintains itself. It is the only logical answer. The truth has been hidden by science for almost 200 years, yet was revealed long ago in the 1800s by scientists such as Antoine Béchamp, who documented in his own experiments that viruses are terrain dependent, non-living agents that break down waste matter, that they come from within, not from without.
Viruses are nothing more than proteins that cleanse. The same is true about cancer. Cancer is another way the body tries to heal itself, by cocooning dead cells in a tumor in which the body is incapable of removing properly so that it can dissolve and cleanse those cells from the body at a later time. The body is miraculous and finds ways to heal no matter the circumstances. It has ways of short-circuiting and short-cutting pathways in times of trouble.
It is sad that modern science has led so many astray in their thinking with regard to their own bodies and how it functions creating nothing but fear and panic, whilst reaping massive amounts of money for those in power as a result. Such fear places a distrust in our own bodies, our neighbors and nature itself, making it appear as if we are powerless in the face of disease; that it is beyond our control and only the medical establishment can save us from ourselves.
How might those in power benefit from such chaos? Explore that thought. This confusion has led to the coronavirus ‘outbreak’ and the resulting fear and chaos which surrounds this manufactured and blown out of proportion event.
This virus is obviously being used to institute police state style laws and measures around the world and these will only increase if the majority do not wake up to the lies surrounding the nature of viruses and disease. References and more at https://www.thebernician.net/the-deception-of-virology-vaccines-why-coronavirus-is-not-contagious-2/
Dr. Shiva Ayadurai says the pharma industry is in peril right now. They’re not making money from pharmaceutical drugs that required enormous investment in R&D, fewer drugs are allowed by FDA because of their toxicity, so pharma companies are moving to vaccines.
Why not, vaccines are pure profit for the pharma industry. There’s no liability, no risk, and especially if they can’t be sued.
Critical thinking
A wide range of posts, transcripts, videos on some of the most important issues of our time. https://www.thebernician.net/critical-thinking/
Evidence of a very dark agenda

Lemons or lemonade?
Individuals can cycle in anger about being deceived or give thanks for higher awareness, the miracle of our body, and summon the courage to preserve our well being and freedom.
If we keep an open mind, the truth can set us free. Please share. Thank you.
Disclaimer:
This information is not intended to provide medical advice, diagnosis, treatment, cure, or prevent any disease. Views expressed here are for educational purposes only.
Doreen A Agostino
Without Prejudice and Without Recourse
http://freetobewealthy.net
Sent via hardwired computer
All wireless turned off to safeguard life
flucausation

About ourgreaterdestiny
Author, Radio Host, Researcher, Editor, and synthesizer of facts, for people to rescue themselves from unawareness, fear, and limitations.
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GOOD COVID WEBSITE:
https://questioningcovid.com/

One article there is called Is Coronavirus Contagious at
https://nourishingtraditions.com/is-coronavirus-contagious/

Amazingly, it starts off by mentioning the Maunder minimum when there were no sunspots in parts of the 17th and 18 centuries. When sunspots returned, there was a pandemic. Miles' paper on the sunspot cycle discussed a connection between the recent solar minimum and the coronavirus, I think, unless he discussed that privately on CuttingThroughTheFog.com. This article discusses connections between the start of telegraphy and other electrical tech over the decades and pandemics. It also claims that viruses are actually exosomes that remove debris from within cells to help them survive. So the pandemic appears to be caused by 5G, which was started in Wuhan and other places where the death toll has been high. The electrical interference causes the splitting of O2, making it hard for victims to breathe. The ventilators do harm instead of good.